The classic cycle of anabolic steroids taken within 24 hours (for example, injectable testosterone esters) necessarily lead to a suppression of LH and products, respectively, to the suppression of testosterone production.
There are 3 ways to avoid this:
1) Avoid permanent use of highly-androgenic steroids.
This can be accomplished, for example, using the oral steroid (whose half-life of several hours), half the dose in the morning (eg. at 9 AM), while the remaining half of the dose at approximately at the afternoon (at 12 AM). Even 100 mg per day of Methandrostenolone can be used in this way with a slight suppression of testosterone production.
This is due to the fact that high eroven exogenous (produced outside of) testosterone is kept for 3-4 hours, but this has not been enough to trigger the lowering of its own testosterone level (probably due to the fact that the pituitary and the hypothalamus does not respond only the current level of androgens, but also on past levels of testosterone and this 4-hour hormonal racing roughly be ignored endocrine system). The problem with this approach is that the effect on the growth of muscle mass is not very good compared to when the steroid in the blood constant.
2) Use the number and type of steroid, which will not significantly inhibit the production of testosterone. Primobolan in a dosage of 200-400 mg per week suitable for this purpose. However, in this case, the results of such a cycle would not be comparable to the more substantial cycle. Esters of testosterone (eg. drugs like Sustanon, Omnadren, testenat, Testosterone Cypionate, etc.) and nandrolone decanoate (Retabolil, Deca Durabolin) significantly inhibit the production of testosterone, even at a dose of 100 mg per week, so that the use of these drugs in low dosage does not make sense: testosterone production will be suppressed and quite unsignificant progress will be achieved.
3) Generally, anti-androgens could be used, but it’s just completely stop the growth results in a mass and strength.
In those cycles where steroids doses are high enough to effectively increase the results seen an interesting thing. During the first 2 weeks of the cycle only the activity of the hypothalamus is suppressed, and it produces much less LHRH as a result of high levels of steroids.
Activity of the pituitary gland at this time is not suppressed at all: in fact, LHRH receptors are sensitive and will respond to LHRH (if it is produced) even more than usual. However, after two weeks, the activity of the pituitary gland is also suppressed, and even if LHRH is produced, the pituitary will produce little or no produce LH. In this case, there comes a deeper type of suppression of the arc. Apparently, after the suppression of this point, there is no next point, where again the suppression becomes deeper. But over time, the restoration is complicated.
Practically there is no distinction between the use of steroids for 3 weeks and 8 weeks: recovery will take the same time. Between 8 and 12 weeks, it becomes increasingly likely that the recovery will be harder and slower, even though the 12-week cycle typically does not create too many problems, and recovery takes only a few weeks. Cycles longer than 12 weeks can create substantial problems with recovery.
It is not known exactly what changes occur in the hypothalamus and pituitary gland when courses are too long, but in practice it appears that the more the cycle goes for 8 weeks, the longer and more difficult the subsequent recovery. There is a suspicion that what occurr violation of the mechanisms of secretion of LHRH hypothalamus.
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